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193 In addition, Fusobacterium nucleatum can activate the RIPK1-mediated apoptosis pathway in UC, resulting in the destruction of tight junctions between cells, which in turn promotes the destruction of the intestinal barrier, inhibits the renewal of intestinal epithelial cells, and accelerates the progression of UC

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The primary mechanisms we see cited in the literature include: VEGF upregulation: BPC-157 increases vascular endothelial growth factor, which drives new capillary formation in injured tissue